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Session Details

＜This site is available for registered users only.＞ （大会参加者専用） Please log in with your participant account. » Participant Log In ＜日本細胞生物学会および日本発生生物学会会員で大会参加者以外の方＞ 要旨閲覧パスワードが必要な場合は、会員番号・氏名・所属機関名を明記の上、合同大会事務局 jscb-jsdb2018@jsdb.jp までメールにてご連絡ください。

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Single-cell imaging analyses in C. elegans revealed a marked temporal difference between chromatin alterations mediated by ASD causative genes and gene expression for neuronal maturation

Mammals such as rodents are most commonly and widely used model organisms to study human diseases and to identify potential therapeutics. However, several experimental approaches using non-mammalian models are also advantageous compensating for the mammalian systems. For some kinds of diseases, non-mammalian organisms can be more suitable models than mammals for the understanding of disease onset at the cellular-level or for a large-scale drug screening. In this symposium, we will focus on recent approaches using non-mammalian organisms including yeast, C. elegans, Drosophila and small fishes that shed light on cellular and/or developmental pathogenesis of various diseases including prion transmission, neurological diseases, autisms and cancer. We would like to discuss their features and advantages in comparison to the mammalian systems in this symposium.

Joint Meeting of JSCB and JSDB

Joint Annual Meeting of 51st JSDB and 70th JSCB

Joint Annual Meeting of 51st JSDB and 70th JSCB

[S03-02]Single-cell imaging analyses in C. elegans revealed a marked temporal difference between chromatin alterations mediated by ASD causative genes and gene expression for neuronal maturation

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