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Pharmacologically increasing Opa1 expression levels by silencing the miR-148/152 family improves the phenotype in mitochondrial myopathy.

Mitochondria integrate cellular signaling and metabolism, thereby determining the fate of organisms. Here, we introduce innovative technologies that unveil previously hidden aspects of mitochondrial biology and pathophysiology, which were obscured by past methodological limitations. The use of newly innovated and developed technologies, such as gene-editing technology, deep-learning, and regenerative engineering, would enable us to overcome the limitations of scientific approaches.<br>

日本分子生物学会年会

The 47th Annual Meeting of the Molecular Biology Society of Japan

The 47th Annual Meeting of the Molecular Biology Society of Japan

[2PS-10-05]Pharmacologically increasing Opa1 expression levels by silencing the miR-148/152 family improves the phenotype in mitochondrial myopathy.

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