Presentation Information
[3O04m1-01]HDAC3 inhibition ameliorates dystrophic axons and memory function via secretion of IGFBP6 from microglia in a transgenic mouse model of Alzheimer's disease
*Tomoharu Kuboyama1, Hikari Okamoto1, Chihiro Tohda2 (1. Lab of Pharmacognosy, Daiichi Univ of Pharmacy, Fukuoka, Japan, 2. Sec of Neuromedical Science, Inst of Natural Medicine, Univ of Toyama, Toyama, Japan)
Keywords:
Alzheimer's disease,microglia,HDAC3,axon
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