Session Details

[1P]69 Alzheimer's Disease and Dementia

Wed. Jul 24, 2024 12:50 PM - 1:50 PM JST
Wed. Jul 24, 2024 3:50 AM - 4:50 AM UTC
Poster Room Marine Messe Fukuoka Hall B

[1P-469]Chronic kidney disease elevating tau phosphorylation in the aged mouse brain

*Tatsuya Manabe1, Takuto Iida1, Takashi Saito1 (1. Dept Neurocognitive Science, Institute of Brain Science, Nagoya City Univ Grad Sch Med Sci, Nagoya, Japan)

[1P-471]Reduced dynamic range and impaired neural coding in 5xFAD mice

*Simon R Schultz1, Mary Ann Go1, Kat E. Clarke2, Yimei Li1, Seigfred Prado3,1, Beatriz R.F. Texeira1 (1. Dept Bioengineering, Imperial College London, London, UK, 2. Dept Biomed Eng, Univ of Melbourne, Melbourne, Australia, 3. Dept Elec Eng, Univ Santo Tomas, Manila, Philippines)

[1P-473]Gene expression profiles of orphan G protein-coupled receptors in Alzheimer’s disease model mouse

*Atsushi Shimohata1,2, Okiru Komine1,2, Akira Sobue1,2, Hiroyuki Mizoguchi3,4, Takashi Saito1,5, Takaomi C Saido6, Naruhiko Sahara7, Makoto Higuchi7, Koji Yamanaka1,2 (1. Department of Neuroscience and Pathobiology, Research Institute of Environmental Medicine, Nagoya University, Aichi, Japan., 2. Department of Neuroscience and Pathobiology, Nagoya University Graduate School of Medicine, Japan., 3. Research Center for Next-Generation Drug Development, Research Institute of Environmental Medicine, Nagoya University, Aichi, Japan., 4. Department of Neuropsychopharmacology and Hospital Pharmacy, Nagoya University Graduate School of Medicine, Aichi, Japan., 5. Department of Neurocognitive Science, Institute of Brain Science, Nagoya City University Graduate School of Medical Sciences, Aichi, Japan., 6. Laboratory for Proteolytic Neuroscience, RIKEN Center for Brain Science, Saitama, Japan., 7. Department of Functional Brain Imaging, National Institute of Radiological Sciences, National Institutes for Quantum and Radiological Science and Technology, Chiba, Japan.)

[1P-475]More severe white matter degeneration within the visual attention network associated with the presence of β-amyloid pathology in mild cognitive impairment

*Yao-Chia Shih1, Yi-Fang Chuang2,3, Yan-Siang Huang4, Sui-Hing Yan4, Yi-Chien Liu5, Shan-Ying Wang6, Yen-Wen Wu6, Yang-Teng Fan1, Elsa Lin-Chin Mai7, Yen-Ling Chiu1,8 (1. Graduate Institute of Medicine, Yuan Ze University, Taoyuan, Taiwan, 2. Institute of Public Health, National Yang Ming Chiao Tung University, Taipei, Taiwan, 3. Department of Psychiatry, Far Eastern Memorial Hospital, New Taipei City, Taiwan, 4. Department of Neurology, Far Eastern Memorial Hospital, New Taipei City, Taiwan, 5. Department of Neurology, Cardinal Tien Hospital, New Taipei City, Taiwan, 6. Department of Nuclear Medicine, Far Eastern Memorial Hospital, New Taipei City, Taiwan, 7. Department of Ophthalmology, Far Eastern Memorial Hospital, New Taipei City, Taiwan, 8. Department of Medical Research, Far Eastern Memorial Hospital, New Taipei City, Taiwan)