セッション詳細
[WS(P)07]Macrophage 1
2024年12月3日(火) 16:50 〜 17:35
Poster 7
[WS07-01-P]Alveolar macrophage-specific depletion system in mice reveals the unique roles in respiratory infections
○Yuki Nakayama1,2, Miwa Sasai1,2,3,4, Masahiro Yamamoto1,2,3,4 (1.Department of Immunoparasitology, Research Institute for Microbial Diseases, Osaka University, Osaka, Japan, 2.Laboratory of Immunoparasitology, WPI Immunology Frontier Research Center, Osaka University, Osaka, Japan, 3.Center for Infectious Disease Education and Research, Osaka University, Osaka, Japan, 4.Center for Advances Modalities and Drug Delivery Systems, Osaka University, Osaka, Japan)
[WS07-02-P]Transcription Factor FOXO1 in Macrophages Regulates Inflammation and Pathogenesis of ARDS in Mouse Model
○Hinata Wade, Masahiro Kitabatake, Ryutaro Furukawa, Atsushi Hara, Noriko Ouji-Sageshima, Toshihiro Ito (Department of Immunology, Nara Medical University)
[WS07-03-P]Lipopolysaccharide pre-conditioning enhances the bactericidal activity of Kupffer cells against both gram-positive and negative bacteria in mice
○Hiroyuki Nakashima, Bradley Michael Kearney, Kazuma Mori, Ryohei Suematsu, Kohei Yamada, Masahiro Nakashima, Manabu Kinoshita (Immunology and Microbiology, National Defense Medical College)
[WS07-04-O/P]The differential pyrin inflammasome responses between resident peritoneal and bone marrow-derived macrophages
○Izumi Sasaki1, Shiori Kaji2, Yuri Fukuda-Ohta1, Daisuke Okuzaki3, Takashi Kato1, Tsuneyasu Kaisho1 (1.Department of Immunology, Institute of Advanced Medicine, Wakayama Medical University, 2.Second Department of Internal Medicine, Wakayama Medical University, 3.WPI-Immunology Frontier Research Center, Osaka University)
[WS07-05-O/P]Clathrin heavy chain: a regulatory key for NLRP3 inflammasome activation via endocytosis in macrophages
○HUNG HIEP HUYNH1, Eri Koike1, Masumi Shimizu1, Akihiko Yoshimura2, Rimpei Morita1 (1.Department of Microbiology and Immunology, Nippon Medical School, 2.Graduate School of Medicine, Keio University)
[WS07-06-P]NLRP3 and SGPL1 interaction plays a key role in priming event for inflammasome activation
○Fumiyuki Sasaki, Masumi Shimizu, Misaki Wakasugi, Hinata Hirashima, Rimpei Morita (Department of Microbiology and Immunology, Nippon Medical School, Tokyo, Japan)
[WS07-07-P]Gelsolin from macrophages promotes fibroblasts migration during skin wound healing
○Eri Toyohara1,2, Fumiyuki Sasaki2, Teruyuki Dohi1, Rei Ogawa1, Rimpei Morita2 (1.Department of Plastic, Reconstructive and Regenerative Surgery, Nippon Medical School, Tokyo, Japan, 2.Department of Microbiology and Immunology, Nippon Medical School, Tokyo, Japan)
[WS07-08-O/P]TAK1-binding protein 2 (TAB2) suppresses aberrant activation of NLRP3 inflammasome mediated by autocrine TNF-α
○Giichi Takaesu1,2,3, Tanveer Ali2, Goro Matsuzaki1,2,3 (1.Tropical Biosphere Research Center, University of the Ryukyus, 2.Department of Host Defense, Graduate School of Medicine, University of the Ryukyus, 3.Advanced Medical Research Center, University of the Ryukyus)
[WS07-09-P]TAK1 is involved in the maintenance of monocyte-derived macrophages that
emerge during the acute phase of inflammation
○Katsuki Iwahori, Hideki Sanjo (Department of Molecular and Cellular Immunology, Shinshu University School of Medicine)
[WS07-10-O/P]A critical role of protein cross-linking enzyme transglutaminase 2 in M2 macrophage polarization and fibrosis
○Hideki Tatsukawa, Kiyotaka Hitomi (Graduate School of Pharmaceutical Sciences, Nagoya University)
[WS07-11-O/P]Autologous Macrophages induced by IL-34-based condition Suppress Hepatic Fibrosis with CD8+ T Cell Inhibition
○Yuichi Igarashi, Haruka Wada, Ken-ichiro Seino (Division of Immunobiology, Institute for Genetic Medicine, Hokkaido University.)
[WS07-12-O/P]Dual-wield pathway of macrophages drives myofibroblast transition via dysregulation of iron metabolism
○Hiroshi Nabeshima1,2, Kiyoharu Fukushima2,3,4, Shizuo Akira2,3,5 (1.Host Defense Laboratory, Immunology Unit, Osaka Research Center for Drug Discovery, Otsuka Pharmaceutical Co., Ltd., 2.Laboratory of Host Defense, World Premier Institute Immunology Frontier Research Center (WPI-IFReC), Osaka University, 3.Department of Host Defense, Research Institute for Microbial Diseases (RIMD), Osaka University, 4.Department of Respiratory Medicine and Clinical Immunology, Osaka University Graduate School of Medicine, 5.Center for Advanced Modalities and DDS (CAMaD), Osaka University)
[WS07-13-P]PDGFRα- fibroblasts and macrophages cooperatively suppress the necrotic changes in myocardial infarction.
○Risa Fujimoto1, Kentaro Fujii2, Masaru Ishii1,2 (1.Department of Immunology and Cell Biology, Graduate School of Frontier Biosciences Osaka University, 2.Department of Immunology and Cell Biology, Graduate School of Medicine, Osaka University)