Presentation Information

[1T07-07]Regulation of the interaction of BCR-ABL with VDAC1 can function as a novel therapeutic strategy against CML.

Yuta Okuda1, Miho Takahashi1, Aoi Ikegami1, Norihito Shibata2, Mikihiko Naito3, Kiyotaka Nishikawa1 (1.Molecular Life Sciences Laboratory, Faculty of Life and Medical Sciences, Doshisha University, 2.Division of Biochemistry, National Institute of Health Sciences, 3.Laboratory of Targeted Protein Degradation, Graduate School of Pharmaceutical Sciences, The University of Tokyo)
キーテクノロジー:BioID法

Keywords:

BCR-ABL、PHドメイン、ミトコンドリア、アポトーシス、VDAC1

cygk2003@mail4.doshisha.ac.jp

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