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The regulations of ATR-dependent tolerance mechanisms in response to oncogenic KRAS-induced replication stress

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Genomic DNA in nucleus and mitochondria must be inherited properly. However, DNA replication is often compromised by obstacles stems from intrinsic and extrinsic stimuli, leading to genome instability and various diseases including cancer. In this symposium, we will discuss the latest findings on the cellular strategy to maintain genome stability or to tolerate genome stress from the aspect of molecular mechanism and cancer biology, focusing on PrimPol.<br>

The 47th Annual Meeting of the Molecular Biology Society of Japan

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[1PS-12-02]The regulations of ATR-dependent tolerance mechanisms in response to oncogenic KRAS-induced replication stress

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