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Towards pathological understanding of neurodegenerative diseases by dipeptide repeats derived from aberrant expansion of C9orf72 with a focus on FG motifs

Frontotemporal lobar degeneration (FTLD) is a type of dementia characteristic of atrophy in the frontal and temporal lobe. FTLD-TDP, featured by a nuclear protein TDP-43 accumulation in the cytoplasm, dominates about a half of FTLD patients. However, it has not been developed to cure FTLD-TDP because of the unknown mechanism. Here we introduce TDP-43 pathology recently reported and novel action of causal proteins of FTLD-TDP, and discuss the mechanism of FTLD-TDP.

【J】Rethinking FTLD-TDP from novel action of causal proteins

The 47th Annual Meeting of the Molecular Biology Society of Japan

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[2MS-07-02]Towards pathological understanding of neurodegenerative diseases by dipeptide repeats derived from aberrant expansion of C9orf72 with a focus on FG motifs

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