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Administration of Nicotinamide Mononucleotide, a Precursor of NAD+, Attenuates Doxorubicin-induced Cardiotoxicity and Renal Injury in Mice.

3. Development and Regeneration - a. Organogenesis / Morphogenesis / Regeneration - b. Stem Cell - c. Cell Differentiation - d. Early Development - e. Reproduction - f. Others 4. Animal and Plant Physiology, and Diseases - a. Symbiotic Microorganism - b. Biological Rhythm - c. Brain / Nervous System / Neurodevelopment / Structure - d. Brain / Nervous System / Behavior - e. Brain / Nervous System / Disorder - f. Immunity - g. Infection / Virus - h. Aging - i. Cancer Cell - j. Cancer Tissue / Cancer Therapeutics - k. Metabolism / Nutrition - l. Genetic Disorder - m. Plant - o. Others

Poster Presentation

The 47th Annual Meeting of the Molecular Biology Society of Japan

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[3P-858]Administration of Nicotinamide Mononucleotide, a Precursor of NAD⁺, Attenuates Doxorubicin-induced Cardiotoxicity and Renal Injury in Mice.

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[3P-858]Administration of Nicotinamide Mononucleotide, a Precursor of NAD+, Attenuates Doxorubicin-induced Cardiotoxicity and Renal Injury in Mice.

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[3P-858]Administration of Nicotinamide Mononucleotide, a Precursor of NAD⁺, Attenuates Doxorubicin-induced Cardiotoxicity and Renal Injury in Mice.