Presentation Information
[2P-052]The dual function of PrimPol switched by ATR is responsible for replication fork stability disturbed by oncogenic KRAS
○Taichi Igarashi1, Yasuhara Takaaki1, Bunsyo Shiotani2 (1. Lab. of Genome Stress Response, Kyoto Univ., 2. Dep. of Genome Stress Signaling, Tokyo Med. Univ.)
Comment
To browse or post comments, you must log in.Log in
