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Tue. Mar 10, 2026

Wed. Mar 11, 2026

Thu. Mar 12, 2026

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Cells adapt to extracellular acidic pH through TM9SF3-mediated PI(4,5)P<sub>2</sub> flop

Recent advances in molecular biology and genetics have highlighted that structural and functional disruptions of organelles—such as mitochondria, lysosomes, the endoplasmic reticulum, and their interactions with the plasma membrane—contribute to a wide range of diseases, including neurodegeneration, cardiovascular dysfunction, metabolic disorders, and immune dysregulation. Increasing attention is also being paid to inter-organelle communication as a key factor in maintaining cellular homeostasis. This symposium aims to promote cross-cutting insights beyond isolated pathologies. It seeks to advance understanding of intracellular processes, encourage new research in this exciting field, and foster the development of future diagnostic and therapeutic strategies.

Decoding intracellular pathology

The 103rd Annual Meeting of The Physiological Society of Japan

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[3S07m-02]Cells adapt to extracellular acidic pH through TM9SF3-mediated PI(4,5)P₂ flop

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[3S07m-02]Cells adapt to extracellular acidic pH through TM9SF3-mediated PI(4,5)P2 flop

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[3S07m-02]Cells adapt to extracellular acidic pH through TM9SF3-mediated PI(4,5)P₂ flop