[3S03a] シナプスの形態と機能の異常がどのように発達障害発症につながるか？

[3S03a-1] シナプス機能におけるNedd4-2を介したユビキチン化の役割

*六本木 麗子¹、獺ヶ口 笑保人¹、Kaplan Öyküm^2,6、Bekir Altas^2,3,4,5、Rhee Jeong²、Brose Nils²、川辺 浩志¹ (1. 群馬大学大学院医学系研究科薬理学分野、2. Department of Molecular Neurobiology, Max Planck Institute for Multidisciplinary Sciences, Göttingen, Germany、3. International Max Planck Research School and the Göttingen Graduate School for Neurosciences, Biophysics and Molecular Biosciences, Göttingen, Germany、4. The Göttingen Graduate School for Neurosciences, Biophysics, and Molecular Biosciences, PhD Program Systems Neuroscience, University of Göttingen, Göttingen, Germany、5. Department of Pharmacology and Program in Neuroscience, University of Maryland School of Medicine, Baltimore, MD, USA、6. The Göttingen Graduate School for Neurosciences, Biophysics, and Molecular Biosciences, PhD Program Molecular Physiology of the Brain, University of Göttingen, Göttingen, Germany)

キーワード：

Nedd4-2、Ubiquitination、synaptic function

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