講演情報
[7a-P05-68]CNT-Modified Transition Metal Dichalcogenide Memristors for Brain-Inspired Nociceptive Emulation
〇(P)Elamaran Durgadevi1, Masahiro Sakai1, Daisuke Kiriya1 (1.The Univ. of Tokyo)
キーワード：
Neuromorphic、Memristor、Nociceptors
A nociceptor is a specialized type of sensory receptor found in neurons that detects harmful or noxious stimuli and quickly alerts the central nervous system to trigger an immediate motor response in both the human body and humanoid robots. In this work, we present a novel memristive device based on a hybrid structure of carbon nanotubes (CNTs) and molybdenum disulfide (MoS2), engineered to mimic the fundamental characteristics of biological nociceptors. The CNT– modified 2D material based memristor exhibits tunable resistance states, strong nonlinearity, and robust hysteresis, enabling the emulation of key nociceptive features such as, threshold, no adaptation and sensitization. In this CNT–2D material-based memristor, carbon nanotubes (CNTs) are sparsely distributed over the 2D material channel and function as localized charge trap sites. When an external bias is applied, carriers become trapped or detrapped at these CNT locations, thereby modulating the local charge distribution in the 2D material (e.g., MoS2, WSe2) and altering the overall device resistance. This interface-driven trapping mechanism allows for controllable, non-volatile switching behavior, without relying on filamentary conduction paths. For device fabrication, 2D material flakes were mechanically exfoliated onto a SiO2 layer on a heavily doped p-type silicon substrate. Two-terminal Ti/Au electrodes were patterned using standard photolithography, followed by metal deposition and lift-off. A diluted CNT solution was then drop-cast onto the 2D material channel for 1 minute and dried using a nitrogen (N2) blow, enabling the formation of localized trap sites. After doping with the diluted CNT solution, the CNTs are well dispersed across the MoS2 channel, serving as effective trap centers that enable memristive hysteresis and facilitate nociceptive behavior emulation.